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As a result, multiple mediator networks were stimulated, such as complements, kinins, coagulation, and fibrinolysis cascades, along with the release of chemokines, cytokines, soluble receptors, lipid mediators, and numerous enzymes such as elastase, myeloperoxidase, and proteases.
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Two multiple mediator models were tested in a sample of 77 undergraduate students.
The parallel multiple mediator model is presented in Figure 1.
No corrections for multiple mediator measurements were performed.
Multiple mediator models in which MHSE is pitted along-side other theoretically relevant mediator variables should be studied.
Figure 1C shows the proposed mediation model with multiple mediators.
The major inflammatory mediators are neutrophils, which release multiple mediators, including cytokines, free radicals, histotoxic enzymes and other proteases.
The likelihood of multiple mediators for knowledge sharing is yet to be investigated.
Cross-lagged multiple regressions and bootstrapping procedures for testing multiple mediators supported all of the relationships proposed by the TPB.
Presence of multiple mediators in the TME and lack of interferon γ (IFNγ) allow activation of TAMs.
The complexity of mediator interdependency may also require the targeting of multiple mediators simultaneously or combined targeting of pro- and anti-inflammatory mediators.
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