Multiple mechanisms contribute to the anti-inflammatory effects of glucocorticoids.
Acquisition of repressive histone marks, like H3K27 methylation, is only one of multiple mechanisms contributing to silencing of tumor suppressor genes in cancer cells [18].
It is likely that multiple mechanisms contribute to PM2.5-exposure induced cardiovascular-related morbidity and mortality.
Multiple mechanisms contribute to arterial disease (e.g. atherogenesis) in patients with type 2 diabetes.
In fact, multiple mechanisms contribute to the development and progression of prostate cancer.
Multiple mechanisms contribute to creating the primary Ig heavy (IgH) and light chain (IgL) diversity.
It is possible that multiple mechanisms contribute to poor fetal growth in the presence of gastroschisis.
Anemia is common following RYGB, and multiple mechanisms contribute to its development in different stages of the clinical setting [ 8].
Multiple mechanisms contribute to apoptosis resistance in SCLC, including elevated expression of the antiapoptotic Bcl-2 protein (Supplementary Figure S1).
Multiple mechanisms contribute to the autocrine ligand-induced EGFR activity [ 31] that is detected in many human tumors [ 32, 33].
