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Lungs in particular are highly sensitive to injury, during the transplantation process generally, and to multiple immune rejection mechanisms.
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In summary, lessons from the panel of animal model systems suggest that multiple elements contribute to the lack of cancer immune rejection.
Despite recent therapeutic success, it is still not common because a large number of transplanted islets get damaged by multiple challenges including instant blood mediated inflammatory reaction, hypoxia/reperfusion injury, inflammatory cytokines, and immune rejection.
But many scientists foresee a problem: immune rejection.
Second, transplant experts expect to face other problems involving immune rejection, although less daunting ones.
The animals are genetically matched, so there is no risk of immune rejection.
This has fostered ongoing investigation into better strategies to manage immune rejection.
Treating patients with cells derived from their own stem cells would avoid any problems of immune rejection.
For therapy, far off as that is, treating patients with their own cells would avoid the problem of immune rejection.
The result was safer and more effective blood transfusions, with less chance for immune rejection of the donor blood.
In this case, there would be no immune rejection [7].
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