Sentence examples for multiple human carcinomas from inspiring English sources

Increased levels of fascin-1 transcript have been reported in multiple human carcinomas (e.g. [10], [13]).

Because fascin up-regulation correlates with poor prognosis and metastatic progression in multiple human carcinomas, fascin is emerging as an attractive potential therapeutic target.

Previous studies have reported that chemical hypoxia can induce strong doxorubicin resistance through HIF-1 upregulation in multiple human carcinomas.

To understand whether REGγ is a tumor-associated protein, we examined REGγ expression levels in multiple human carcinomas.

The humanized version of PankoMab has been shown to react to the tumor expressed MUC-1 in multiple human carcinomas, although no clinical trials have been published [ 42].

We demonstrated that TCRγ9δ2 OT3 -Fc, a fusion proTCRγ9δ2 OT3 -Fcf the complete extracellular domains ofusionγ9 and δ2 chains linked to the Fc domains of human IgG1, exhibited successful binding to multiproteinan composeda cell lines.

We conclude that simultaneous analysis of multiple polypeptides in human carcinomas can be achieved by 2-DE and may be useful in prognostic studies, and that malignant progression of breast carcinomas results in the decreased expression of cytokeratin polypeptides.

Our previous studies have shown that Brachyury is aberrantly re-expressed in multiple types of human carcinomas and that high levels of Brachyury expression drive the phenotypic conversion of tumor cells into mesenchymal-like cancer cells.

Consistently, SAG was found to be overexpressed in multiple human cancer tissues including carcinomas of the lung, colon, stomach, and liver, and SAG overexpression correlates with poor survival of lung cancer patients.

On the other hand, it has been shown that mutationally enhanced activity of c-RET kinase caused the development of human carcinomas, including multiple endocrine neoplasia (MEN) and papillary thyroid carcinoma (PTC) [4], [8].

Fascin is an actin-bundling protein that is absent from most normal epithelia yet is upregulated in multiple forms of human carcinoma, where its expression correlates clinically with a poor prognosis.