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Adhesion of meningococci on hCMEC/D3 monolayers induces translocation of multiple endothelial membrane proteins, including ezrin, moesin, and actin to form honeycomb cortical plaques beneath the meningococcal colonies.
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Presumably, the large molecular size of the dissolved molecules limited their ability to cross the endothelial membrane.
Endothelial tubulogenesis is a complex process that requires multiple endothelial cell functions, including proliferation, migration, invasion and differentiation [16].
It is also present in the luminal endothelial membrane.
Blood vascular islands often contained multiple endothelial cells.
These models show that epithelial endothelial binding occurs rapidly and that trans-endothelial migration is initiated at the intercellular joints between multiple endothelial cells.
Black arrows show close contact between infected red blood cells and the endothelial membrane.
This suggests alteration of pulmonary endothelial membrane transport properties due to O2 toxicity.
MMP9 is released from granules of neutrophils and induces capillary leakage by degrading endothelial membranes.
After Nf1 loss or HRasG12V expression, TJs were present between endothelial membranes but contained gaps.
It also solubilizes lipids of endothelial cell membrane resulting in membrane fluidization which in turn opens the tight junction between endothelial cells.
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CEO of Professional Science Editing for Scientists @ prosciediting.com