Sentence examples for multiple adhesion molecules from inspiring English sources

Angiogenesis also depends on adhesive interactions between vascular cells of which multiple adhesion molecules are involved, such as αv β3, αv β5, α5 β1, and α2 β1 integrins; PECAM-1; and VE-cadherin (Heimark et al, 1990; Lampugnani et al, 1991, 1992; Brooks, 1996).

Multiple adhesion molecules and their regulators are expressed at different levels between hESC and phESC derivates.

Migration of lymphoid cells throughout the body is regulated by the expression of multiple adhesion molecules and chemokine receptors that interact with corresponding ligands on endothelial and stromal cells.

It is likely that multiple adhesion molecules are involved in leukocyte infiltration into brain after ICH.

Multiple adhesion molecule pathways appear to mediate leukocyte interactions, particularly rolling, within the synovial vasculature.

However, we failed to observe the M1 axon defects in multiple adhesion molecule mutants, including fmi-1 and mutations of other IgCAM genes that are expressed in the pharynx and that could potentially mediate the M1/g1P fasciculation.

The most studied example is endothelial targeting via multiple cell adhesion molecules (CAMs), which mimics the strategy of leukocytes to adhere and traverse the vascular endothelium.

Being a multidomain matrix glycoprotein capable of interacting with multiple cell adhesion molecules and proteases involved in angiogenesis, it is not surprising that THBS1 plays important roles in the development of diverse tissues [ 118].

Nissen et al. [ 24] studied the serum levels of multiple cell adhesion molecules in aneurysmal SAH patients with and without delayed ischemic neurological deficit (DIND) and did not find differences in the levels of ICAM-1, VCAM-1, platelet endothelial cell adhesion molecule-1 (PECAM-1), or E-selectin between the two cohorts.

Control of T cell activation and modulation of T cell function not only depend on the TCR-epitope-MHC complex interaction, but also on a collective process that involves multiple adhesion and regulatory molecules spatially organized at the T cell-APC interface, forming the T cell IS (Fooksman et al., 2010).

In the field of basic immunology, T cell efficacy is not only controlled by the specificity and avidity of the tumor antigen and T cell interaction, but it also depends on a collective process, involving multiple adhesion and regulatory molecules, spatially organized at the T cell IS.