Sentence examples for multiple abnormalities that from inspiring English sources

Cardiovascular reflex testing reveals multiple abnormalities that reflect probable disturbance of parasympathetic/sympathetic balance.

Noonan syndrome is a congenital syndrome characteristic of multiple abnormalities that include facial dysmorphology and cleft palate, short stature, and hypertrophic cardiomyopathic defects that include defects in the vascular smooth muscle architecture [ 137].

Thalassemic patients present with multiple immune abnormalities that may predispose them to oral Candida, however this has not been investigated.

There are multiple sonographic abnormalities that may be seen with congenital toxoplasmosis; however, these are only suggestive of fetal infection.

The majority of breast cancers involve multiple molecular abnormalities that are likely to be involved in malignant progression.

This situation could be clinically relevant, as cancer cells contain multiple molecular abnormalities that require therapeutic correction in order to restore the desired phenotype.

In this study, exome sequencing was performed on a couple and from samples from their three affected fetuses, all of whom had been terminated on account of multiple congenital abnormalities that included PMG.

Recently, in the same cohort, the association is found strongest among subjects with multiple molecular abnormalities that confer the greatest risk of progressing to cancer, but absent in those at the lowest risk (Galipeau et al, 2007).

Together, these findings indicate that the breast cancer of African American women contains multiple biological abnormalities that may represent a different mutational landscape of breast cancer than in Caucasian women, and that is associated with accelerated proliferation and growth, and could play an important role in the earlier appearance of breast cancer in African American women.

Despite the multiple accumulated genetic abnormalities that cause malignant transformation, however, it is evident that some of the transformed cells can still respond to signals from their external environment.

How autoreactive GC B cells escape this 'deletion by neglect' is not entirely clear, but could involve multiple mechanisms - including, for example, abnormalities that lead to excessive accumulation of CD4 T-follicular helper cells in GCs [ 32] and B-cell intrinsic dysregulation of prosurvival factors in mice containing the Sle1 locus [ 33].