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Multifactorial models suggest that the genetic susceptibility due to specific variant alleles in polymorphisms may affect the outcomes of environmental exposure [ 7].
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Although this coxib effect is probably multifactorial, abundant data from experimental models suggest that it derives at in part from suppressed production of prostacyclin (PGI2), especially relative to that of thromboxane (Tx) A2.
Further, while animal models suggest that the pathogenesis of diabetic gastroparesis is likely to be multifactorial involving the vagus, intrinsic nerves, interstitial cells and smooth muscle of the stomach, the relative importance of these elements in explaining variations in the clinical presentation and natural history of patients remains unknown [ 2].
Models suggest it could.
Some models suggest there could well be.
Our toy models suggest three things.
However, Radloff (1977) argues against undue emphasis on separate factors and suggests using a simple total score to measure depressive symptomatology, so multifactorial models could be more justified if they include a higher order construct.
This model suggests that the gastric oncogenesis dynamic implies a multifactorial and sequential progression, apparently slow, of chronic superficial gastritis (SG) to premalignant lesions such as chronic atrophic gastritis, intestinal metaplasia, dysplasia, and finally GC [ 4, 5].
Newer statistical techniques (multifactorial models) can improve precision by incorporating several factors that are related directly to labor progress.
In our multifactorial models, we also observed that the association between sex and head injury corollaries varied by injury mechanism.
In multifactorial models this association was no longer significant.
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