Increased oral mucus was observed in the infected chickens (Sun et al., 2010b).
After high oxygen treatment heavy and loud breathing was noticed in pups during handling, however, no mucus was observed in the airways after mucin staining of lung sections.
22 23 When colon tissue sections from these mice were analysed, essentially normal mucus was observed with regard to thickness and stratified appearance, but it was penetrated by bacteria (figure 1 and online supplementary figure S1A, Slc9a3−/−).
Although increases in mucus and mucin were observed in stimulated conditions, neither increase was statistically significant.
Indeed, mucus plugs were observed in the airways of some mice following exposure to HDM (data not shown).
The patient reported marked improvement of his dyspnea and fewer difficulties with expectoration; no further occurrences of intermittent worsening of dyspnea due to mucus stagnation were observed.
Pathologically, peribronchial infiltration of inflammatory cells, epithelial hyperplasia, and increased mucus production were observed in Il7r−/− mice reconstituted with ILC2s and exposed to Alternaria (Fig. 2C).
Infected ducks usually began showing apparent symptoms on day 3 PI, though their elevated body temperature, excessively excreted oral mucus, and dried cloaca were observed as early as 1 day (24 h) PI when they were swabbed.
Inflammatory cell recruitment and mucus secretion in lung tissue were observed via microscopic examination.
The major dose-related clinical signs included soft/liquid feces and mucus in the feces; these were observed in 0, 0, 1 and 14 does in the 0, 20, 100 and 500 mg/kg/day groups, respectively.
