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CYP-1 is functionalized with sulfate groups to increase water solubility and decrease aggregation where CYP-2 is much more cell permeable and was used for detecting Cd2+ in HeLa cells.
F-FDG treatment also caused much more cell death through the necrotic pathway in large (more than 1 cm) tumors in 22-week-old mice than occurred in untreated tumors (Fig. 2d,2d).
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So in the second week there are much more cells that can be induced to accumulate more HIF-1α, and hence more tremendous outcome in vessel outgrowth.
While only a few tumour cells of PAC120 expressed MUC2, the HID variants contained much more cells staining positive for MUC2.
The result showed that cells treated with YM155 50nM and 100nM for 6 and 12 hours, much more cells showed apoptotic feature compared with control group.
This is another key factor in the genetic regulation of adaptation to hypoxia, allowing programmed cell death (apoptosis) as opposed to the much more harmful cell necrosis.
Schwannoma cells re-expressing SOX10, when viewed by fluorescence microscopy, showed a much more regular cell shape with fewer filopodia and appearing to grow over each other less than GFP control cells.
Moreover, after the laser irradiation, the cells cultured with nrGO-PEG presented much more dead cells compared with the cells cultured with nGO-PEG (Figure 7C).
Statistical analysis showed much more prominent cell processes outgrowth after MSCs conditioned media induction in U251 cells, as is shown in Figure 9.
We conclude that topoisomerase IIalpha is a strict proliferation marker in normal and neoplastic cells in vivo, but that topoisomerase IIbeta has a much more general cell and tissue distribution than has topoisomerase IIalpha.
The TUNEL assay showed that combination treatment induced much more tumor cell apoptosis than individual drugs.
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