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Our study revealed that seminomatous TGCTs are characterized by much higher staining of syndecan-4 in tumour cells compared to NSGCTs.
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In CD sections there was much higher intensity staining of the villous epithelium and the staining exhibited altered distribution throughout these cells (Figure 4B, 4C).
The same picture was observed when CYP1A was localized within the MI tissue by ISH; exposed fish had a much higher CYP1A staining compared to the control fish (only weak and random CYP1A staining).
HUVEC showed a much higher Cx37 staining in areas that adjoined or overlapped with SMC (A) than HUVEC of the same culture which occasionally formed islands of "pure" EC (B).
Because there was a much higher level of staining in deeper laminae, in many cases these cell bodies could only be identified with an oil-immersion lens.
IL-2 treated PBMCs triggered much higher levels of ALP staining when compared to untreated PBMCs (Figs. 5C and 5D).
Staining of delignified tissue samples with FITC-conjugated X-2 resulted in much higher signals in comparison to staining of mature (normal) birch and pine tissues.
Interestingly we noticed that SDF-1 staining was much higher in papillary dermal cells, which reside in the superficial dermis, than in reticular dermal cells.
As seen in Fig. 4, intensity of staining was much higher in the outer cellular cortex layer containing neuronal and glial cells when compared with neuropil areas.
EM of heterogeneous particles preserved in negative staining produces much higher signal-to-noise ratio than unstained cryo-EM.
Conversely, we found much higher levels of pAKT(Ser473) staining in papillomas of the UroIICre + K-Ras G12D/+ Pten fl/+ mice in comparison to UroIICre + Fgfr3 +/K644E K-Ras G12D/+ mice (n=3, P<0.001; supplementary material Fig. S5G,H,K,L).
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