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At maximum flash intensity, the a-wave measure of photoreceptor hyperpolarisation has amplitudes of −28±5.8 µV or −31±3.5 µV, and implicit times of 61±7.6 milliseconds (ms) or 68±7.7 ms in control and drug-treated larvae, respectively.
Analysis of MEG data with respect to tactile CS revealed a well-known SEF sequence [26] with peaks at 39.7±1.8, 75.4±6.8, 117.7±4.6 and 120.3±6.5 ms in control subjects.
Similarly, the b- wave which reflects signal transmission to bipolar cells has amplitudes of 199±20.5 µV or 208±28 µV and implicit times of 157±10.5 ms or 169±10.1 ms in control and drug-treated larvae, respectively (Fig. 7A C).
ERP was 71.5 ± 8.9 ms in control and 72.3 ± 9.3 ms in 30 μM carbenoxolone (no significant difference, n = 8, Figure 5 B).
The mean standing RR intervals in pSS patients was 688 ms (95% confidence interval 670 ms to 755 ms), as compared with 781 ms (95% confidence interval 735 ms to 828 ms) in control individuals.
When intra-atrial pressure was raised to 20 cmH2O, the ERP was 33.3 ± 3.2 ms in control and 35.3 ± 4.7 ms in the presence of 30 μM carbenoxolone (no significant difference, n = 8, Figure 5 B).
Similar(46)
The APD90 was 516.5±26.1 ms in LQT2-specific cardiomyocytes compared with 310.5±19.6 ms in control-iPSC-derived cardiomyocytes or 338.6±19.9 ms in hESC-derived cardiomyocytes.
In transgenic mice the duration of the CAP was longer (6 ms in mutants vs. 3 ms in controls) signifying a decrease in conduction velocity, consistent with hypomyelination.
Remarkably, while the exocytotic response saturated at 90 100 ms in controls, the response did not show saturation in latrunculin-treated IHCs.
The P3a component that occurs around 350 ms in controls was all but absent in the PM groups except in the older children, and the Nc component showed less differentiation among the visual stimuli in the unexposed compared to exposed group.
Mean roofed living area per member of the family was 25.8 square meters in case versus 25 m in control group without statistically significant difference.
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