Sentence examples for ms at baseline from inspiring English sources

Broad maximum P waves (≥120 ms) at baseline were observed in 70% of patients with AF and 41% of controls (p = 0.002).

nNOS blockade also resulted in lowering of the left (28 ± 3 vs 54 ± 3 ms at baseline; P =.0002, Tukey-Kramer) and right (45 ± 6 vs 60 ± 0 ms at baseline; P =.001, Tukey-Kramer) VT thresholds.

Addition of systemic nNOS blockade was associated with a reduction of both left (40 ± 2 vs 49 ± 2 ms at baseline; P =.001, Tukey-Kramer) and right (41 ± 2 vs 49 ± 2 ms at baseline; P =.03, Tukey-Kramer) vERP.

In conditions of beta-adrenoceptor blockade (atenolol), atropine administration reduced AVNERP (62 ± 6 vs 77 ± 3 ms at baseline; P =.003, Tukey-Kramer) and SNRT (171 ± 7 vs 219 ± 5 ms at baseline; P =.002, Tukey-Kramer) and lowered the S1 coupling interval required to achieve 2 1 heart block (64 ± 1 vs 73 ± 2 ms at baseline; P =.0008, Tukey-Kramer).

Measurements obtained during the vERP assessment protocol also revealed QT prolongation in conditions of DVMN inhibition (60 ± 4 vs 52 ± 2 ms at baseline; P =.04, Tukey-Kramer).

QT dispersion decreased from 63 ± 24 ms at baseline to 54 ± 16 ms (P = 0.3) at 2 months of follow-up.

In the same sitting, SICI was assessed at ISI of 3 ms. At baseline, SICI at ISI 3 ms was 16.8 ± 1.7% and was significantly greater than SICI at ISI 1 ms (P < 0.05).

The median M at baseline for the centers was 10.6 and 10.0 mgmin−1 lbm−1, respectively.

Only 439 individuals (0.3%) had eGFR <30 mL/min/1.73 m at baseline.

However, two patients had an eGFR <60 mL/min/1.73 m (44 and 53 mL/min/1.73 m) at baseline.

Approximately one-third of PLHIV at both sites had eGFR < 90 ml/min/1.73 m at baseline (Table  2).