Sentence examples for mouse we show from inspiring English sources

Using an ME-deficient mouse, we show that ME is required for MLL-AF9-induced transformation both in vitro and in vivo.

In the Akita mouse we show that in the setting of ER stress, short telomeres cause additive injury that results in loss of β-cell mass.

Using an isoform-specific knockout mouse, we show that hair cells expressing only the small isoform remarkably develop normal stereocilia bundles.

By fluorescently labeling cells that express proglucagon in a transgenic mouse, we show that primary L cells are electrically excitable and directly nutrient responsive.

Using a melanoma cell line derived from a cutaneous nodule of a RET mouse, we show that HB-19 can affect several criteria implicated in the tumorigenic potential of melanoma cells, such as restoration of contact inhibition in culture, reduction of colony formation in soft agar, and impairment of tumorigenicity and lung metastasis in mice.

In mouse, we showed a decreased release probability of excitatory synapses at the hippocampal to basolateral amygdala connections.

Using the HLA-DR4-IE transgenic mouse, we showed that both T-cell proliferation and Th1 cytokine production (IFN-γ) correlate with the predicted affinity of a peptide for DR4.

By crossing ENTPD1-null mice with db mice, we show that ENTPD1 deletion has prominent effects on metabolic syndrome traits.

By engineering TGF-β and BMP-reporter mice, we show that TGF-β2 signaling antagonizes BMP signaling in HFSCs but not through competition for limiting Smad4-coactivator.

Functional outcome was assessed with the horizontal ladder test.Using NSC isolated from CCR2+/+ and CCR2-/ mice, we show that receptor deficiency significantly impaired transendothelial diapedesis specifically in response to CCL2.

Using natural killer T (NKT) cell-deficient mice, we show here that allergen-induced airway hyperreactivity (AHR), a cardinal feature of asthma, does not develop in the absence of V alpha 14i NKT cells.