Sentence examples for mouse we report from inspiring English sources

Using new cardiac-selective ankyrin-G mice (conditional knock-out mouse), we report that ankyrin-G targets Nav1.5 and its regulatory protein calcium/calmodulin-dependent kinase II to the intercalated disc.

The binary transgenic mouse we report here presents an opportunity for such measurements.

Using 3D, time-lapse intravital imaging for direct visualization of the muscle regeneration process in live mice, we report that extracellular matrix remnants from injured skeletal muscle fibers, "ghost fibers," govern muscle stem/progenitor cell behaviors during proportional regeneration.

By using intracerebral inoculation of concentrated urine in cervid PrP transgenic mice, we report the presence of infectious prions in urine from CWD-infected cervids, and confirm the phenomenon of prionsialia in these animals.

In neonatal mice, we report that the PBf response to acidosis is abolished by pre-treatment with the SERT blocker fluoxetine and that SERT mRNA and 5-HT neurons are found in the vicinity of the RTN/pFRG area.

In α-syn transgenic mice, we report prominent mRNA downregulation of 14-3-3 14-3-3 14-3-3 θ in the cortex, a site of intense α-syn pathology.

Here, using LPCAT3-deficient cells and mice, we report that LPCAT3 is critical and relatively specific for the regulation of arachidonate levels in membrane phospholipids in vivo.

As proof of principle, Frech and coworkers [ 58] developed transgenic mice that overexpress ER-α in a C57BL/6 background, in which the number of ER-α positive cells in transfected glands was similar to that in the virgin BALB/c mice we report on here.

Using 2-photon calcium imaging in the ARC of POMC-EGFP mice, we report that a 5-HT2CR agonist was equally effective in stimulating intracellular calcium mobilization in young and middle-aged mice, which suggests that there is no inherent defect in components of the intracellular signaling cascade lying downstream of the 5-HT2CR.

Using transformed primary fibroblasts established from E1A/Ha-Ras-transfected knockout mice, we report here that p53-deficient cells are sensitised to some anticancer agents by the additional loss of the Pms2 gene, indicating that Pms2 positively regulates cell survival by a p53-independent mechanism.

In our maternally protein restricted mice we reported previously that PLP mice remained smaller throughout life whereas recuperated mice remained heavier than control animals throughout most of the adult life [32].