Sentence examples for mouse we determined from inspiring English sources

For both human and mouse, we determined a set of 6-mer motifs exhibiting location-specific overrepresentation using the Functional Region Evaluation Engine (FREE) (Yokoyama et al. 2009).

By scoring the presence of binding sites in all promoters in mouse, we determined the statistical enrichment of GO terms for the putative targets of the applicable group of TFs; note that the set of enriched GO terms are those associated with the putative targets, not those associated with the TFs themselves.

Using two-photon intravital ratiometric CFP/YFP analysis of fMLP-stimulated LFA-1 FRET and Mac-1 FRET mice, we determined the conformational status of LFA-1 and Mac-1 at the trailing edge during uropod elongation.

By assessing engraftment in NOD/SCID/IL2Rγ-null mice, we determined that HSC function resides predominantly in the TIM3-negative fraction of normal bone marrow, whereas LSC function from multiple AML specimens resides predominantly in the TIM3-positive compartment.

Using Foxp3-GFP-KI, LFA-1-KO and Rap1A-GTP-Tg mice we determined that Rap1A has an active role in the development of thymic Treg but LFA-1 is not mandatory for this function.

In a cross of TG with Fgf21-knockout (FGF21 mice, we determined the functional role of FGF21 as a muscle stress-induced myokine under low and high fat feeding conditions.Here we uncovered that FGF21 signaling is dispensable for metabolic improvements evoked by compromised mitochondrial function in skeletal muscle.

Furthermore, using knock-out mice, we determined that the ζ isoform of DGK (DGKζ) is necessary for the mechanically induced increase in PA.

To determine if alterations in the concentration of brain monoamines played a role in the effect of clorg/parg on food intake in mice, we determined the concentration of serotonin, dopamine, and norepinephrine in their cerebral cortex and hypothalamus after clorg/parg administration.

In colonic IEC from germ free mice and matched conventional mice we determined the expression level of nine genes strongly affected by our antibiotics by gavage treatment.

To investigate the phenotypes of C57BL/6J patDp/+ mice, we determined the circadian rhythms of locomotor activity in these mice compared with WT controls.

For further confirmation of normal nociceptive responses in Atxn2 deficient mice, we determined responses to pain stimuli in the hot plate paradigm.