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In order to further examine the difference of modules between human and mouse, we compared the pair-wise modules derived from human and mouse with the use of similarity measurement calculated by Eqa.

To test the significance of this probe distribution in monkey and mouse we compared the proportion of CpG loci that reside in each structure to the total number of CpG loci interrogated on the DNA methylation panel via permutation testing.

To assess whether the H3K27me3/H3K36me3 gene-body chromatin signature is correlated with MAE in mouse, we compared results from a ChIP-Seq analysis (the "chromatin signature") and a traditional analysis of allelic expression bias in clonal cell lines.

In the critically ill mouse, we compared the effect of parenteral nutrition (n = 13) with fasting (n = 11) on body composition, adipocyte cell size, and macrophage accumulation in adipose tissue, liver and lung.

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Using NMRI mice we compared the effects of immunisation with a combination of PA- and BclA-encoding plasmid DNA, to immunisations with either PA- or BclA-encoding pDNA.

Using a recently described skin-humanized model based on the engraftment of human bioengineered skin equivalents onto immunodeficient mice, we compared the efficacy of different in vivo gene transfer strategies aimed at delivering growth factors to promote skin wound healing.

To test the phenotypic differences between M-WT mice and C57 BL/6 mice, we compared their grooming and nociceptive behaviors.

To further characterize structural differences between DAT KO and WT mice we compared FA and TrD images of the two genotypes using statistical parametric maps.

To verify that protein expression was reduced in the heterozygous mice, we compared the relative expression levels of Lck and Fyn in immature and mature CD4+ lineage cells from Lck+/− and Fyn+/− mice with those from WT mice.

To examine whether growth retardation contributes to the body weight phenotype of CXCL14−/− mice, we compared concentrations of growth hormone and IGF-I in the circulation of CXCL14−/− and CXCL14+/− mice, and of CXCL14−/−ob/ob and CXCL14+/−ob/ob mice.

In a separate group of mice, we compared the loss of force in EDL muscles of adult and aged mice immediately following a mild eccentric contraction protocol of 15% strain (Figure 7).

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