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In addition, Muc19, a single-copy gene in human and mouse, was demonstrated to be a putative ortholog of spiggin.
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Cellular and humoral immunogenicity in mice was demonstrated using a DNA prime/rFPV boost and vaccinia virus challenge model.
Inhibition of the effect of hypergastrinaemia on the Shh pathway, in cyclopamine-treated mice, was demonstrated by use of lansoprazole which concomitantly inhibited Gli-1, and did not increase Shh production.
Serial in vivo tumor transplantability in nude mice was demonstrated using H1299 cell line.
infection of mice was demonstrated recently to serve as a model for studying the development of primary pneumonic plague [3].
The inducible Cre activity of the R26-CreERT transgenic mice was demonstrated in neuronal tissues by Badea et al. [27].
Furthermore, MEF2 factors have been shown to mediate the Ca2+/Calcineurin activation of PGC-1α promoter and deletion of MEF2A in mice was demonstrated to result in perturbation of mitochondrial structure[17], [29].
The absence of MMP19 protein in MMP19-deficient (MMP19−/−) mice was demonstrated by immunoblotting of cell lysates prepared from MMP19−/−, heterozygous (MMP19+/−), and wild-type primary keratinocytes (Fig. 1D).
The preponderance of Lf1 in the faecal Lactobacillus isolates in the faecal samples of mice was demonstrated by PCR and RAPD.
The survival of mice was demonstrated using the Kaplan Meier method and the logrank (Cox Mantel) statistical test was applied to compare the curves for non-parametric procedures.
Iron-dependent enhancement of experimental infections in mice was demonstrated with the related bovine parasite Tritrichomonas foetus (Kulda et al. 1999).
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