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30 min later, thiopental sodium (40 mg/kg, i.p). was administered to each mouse to induce sleep.

In order to investigate whether the induced M IL-23) cells could proM IL-23e pathogenesis in the IMQ-inducellssoriasis mouse model, we used a suboptimal dose of IMQ (about 35 mg per mouse) to inducouldweak promoteis-like dermatheis.

Thirty minutes later, thiopental sodium (40 mg/kg, i.p). was administered to each mouse to induce sleep.

In brief, the mTomato/mEGFP reporter gene mouse was bred with NPHS2-Cre mouse to induce a Cre-recombinase-dependent florescence switching from red (mTomato) to green (mEGFP) fluorescence in podocytes.

Test groups and control received respective treatments at proper doses as mentioned earlier, while the positive control group received caffeine at the dose of 20 mg/kg, i.p.. Thirty minutes later, pentobarbitone (50 mg/kg, i.p). was administered to each mouse to induce sleep.

Test groups and control received respective treatments at the proper doses, while the positive control group received diclofenac sodium (25 mg/kg, p.o).. Thirty minutes later, acetic acid (0.7%, 10 ml/kg, i.p). was administered to each mouse to induce abdominal contraction known as writhing [ 20].

Similar(54)

Mouse breast cancer 4T1 cells were injected into the mammary fat pads of mice to induce primary breast tumor growth.

After one hour of administration of drug and extract, 0.6% glacial acetic acid (10 ml/kg) was given i.p. to all the mice to induce pain [13].

The most potent chemical carcinogens require only a single injection in young mice to induce a high incidence of lymphomas, often thymic T-cell lymphoblastic lymphomas.

A single dose of freshly prepared STZ (180 mg/kg body wt). in cold 0.01 M citrate buffer (pH 4.5) was administered intraperitonially to overnight fasted mice to induce diabetes [15].

To study if Mecp2 silencing in intestinal epithelium was able to reproduce the phenotype observed in Mecp2-null mice, transgenic animals expressing Cre recombinase under the control of the Villin promoter (Vil-cre) were crossed with Mecp2 flox/y female mice to induce the deletion of loxP-flanked exon 3 and 4 of Mecp2.

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