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On the other hand, there is compelling data from the mouse study suggesting the potentiality of "Th9" cells in inducing colitis and peripheral neuritis [8].
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A new mouse study suggests it may help relieve one of the miseries of diabetes, a chronic condition in which the stomach fails to empty after a meal.
This mouse study suggests that loss of both p53 and RB is necessary for progression of UCC, but insufficient to initiate invasive UCC.
The overlap of enriched GO terms between human study and mouse study suggests that immune response plays a major role in ankylosing spondylitis development and therefore deserves further studies.
Additionally, mouse studies suggesting associations between high levels of cytokines and CM [ 58- 61] have been recently challenged by works showing that high levels of pro-inflammatory cytokines such as TNF-α are poor indicators of human CM in African children [ 64- 67].
Knockout mouse studies suggest that ablation of one of the several K6 genes can be tolerated owing to compensatory expression of the others.
Recent in vivo mouse studies suggest that WNT7A is an inducer of ovarian cancer growth [ 19].
In addition, recent data from mouse studies suggest that BPA alters gene expression in the placenta (Susiarjo et al. 2013).
Initial data from mouse studies suggest that prion protein is required for synaptic transmission and mice lacking PrPC show reduced excitatory and inhibitory synaptic currents.
On first examination, however, these mouse studies suggest that exposure to airborne mold toxins may adversely affect people's ability to smell.
(Kane, et al, unpublished data, 2014) The transgenic mouse studies suggest that APP signaling can be manipulated to inhibit AD pathophysiology.
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