Now, a new mouse study indicates that the belief that a mother's diet can influence a baby's sex is not so far-fetched.
In conclusion, our mouse study indicates that proximal tubular reabsorption of urine hepcidin-1 occurs in a megalin-dependent manner.
Our study, taken together with the Xcat mouse study, indicates that the unusually large intron 1 of the NHS is important for gene regulation, and that the Xcat mouse is a model for X-linked cataract.
An additional mouse study indicated that nestin-positive CSCs localized to the perivascular niche of medulloblastoma tumor tissue and exhibited radioresistance through the activation of the AKT/PI3K and p53 signaling pathways.
A recent mouse study indicated that the tetracycline derivative minocycline might be an effective therapy for FXS through its ability to inhibit upregulated matrix metalloproteinase-9 (MMP9), a secreted gelatinase that cleaves extracellular proteins to remodel the extracellular matrix.
These mouse studies indicate that dysregulation of postmeiotic repression can lead to male subfertility or infertility.
Mouse studies indicate that either is sufficient for melanoma predisposition and that they might, in fact, cooperate to drive disease progression [76] [77].
Transgenic mouse studies indicate that DAT is critical in maintaining normal dopaminergic neurotransmission and synaptic tone, and that even a 50% reduction in DAT protein (DAT +/− mice) is sufficient to significantly alter DA signaling and synaptic stores [6], [7].
While there are no known human disorders caused by defects in RECQ1 or RECQ5, cellular and transgenic mouse studies indicate that loss of these proteins also leads to genomic instability [5].
Parallel mouse studies indicate that the main effects are due to nab-paclitaxel and not gemcitabine.
