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No statistical difference in the average daily food consumption between mouse strains was observed.
However, the stimulation index obtained with the counts of BrdU-incorporated LNCs with 7-AAD using flow cytometry yielded comparable results and 100% concordance between the BALB/c and CBA/J mouse strains was achieved, suggesting that the performance of LLNA: BrdU-FCM using BALB/c mice was equivalent to that with CBA/J mice.
In our case, the group identity of the three mouse strains was used in the Y-table.
Bleomycin impact on two mouse strains was significantly different as evaluated by survival and histopathological analysis, as previously described [42].
Isolation of CD8+, CD4+CD25+, and CD4+CD25- cells from other mouse strains was performed using MACS microbead separation kits (Miltenyi Biotec Inc., Auburn, CA).
Genomic DNA from inbred mouse strains was purchased from the Jackson Laboratory (Bar Harbor, ME) or was obtained as a gift.
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Much of the variation in survival time between mouse strains is attributable to differences in inherited susceptibility to specific diseases.
The brains of different mouse strains were collected from adult (4 month old mice) or newborn mice.
Best to our knowledge, only inbred [49,51,53,58,59] but not outbred mouse strains were shown to display patient-like AKI alterations.
Some of these mutant mouse strains were found to exhibit behavioral abnormalities reminiscent of human psychiatric disorders.
It has been shown that certain mouse strains are the most sensitive animals for immune modulatory effects of PFOA and PFOS.
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