Sentence examples for mouse strains suggest from inspiring English sources

Strain-dependent differences in vaginal opening among inbred mouse strains suggest that genetic background contribute significantly to the puberty timing, although the exact mechanism remains unknown.

Our results, obtained in two treatment protocols on two mouse strains, suggest an antidepressant-like effect of used here dosing with T2.

Also, experiments in lupus-prone mouse strains suggest that the capacity of macrophages to internalise apoptotic cells is normal when mice are not in a state of active disease [ 30, 31].

In the current study, analyses of combinations of nested and contiguous subcongenic mouse strains suggest that at least two additional modifier genes underlie the Psl1.1 locus and that at least four modifier genes, including Gsta4, underlie the Psl1.2 locus, indicating that Psl1 is a compound quantitative trait locus (QTL).

Additional experiments with several other mouse strains suggested that a gene called Ets2--whose human counterpart is triplicated in people with Down syndrome--accounts for much of the protection.

These expression profiles were observed in six individuals from three mouse strains, suggesting the pattern is not randomly determined.

Initial studies of human SLE patients and lupus-prone mouse strains suggested that B cell depletion usually given together with CY ameliorates disease activity in a subset of patients [9], [10], but two large randomized, placebo controlled studies of B cell depletion with anti-CD20 antibody failed to show efficacy at 12 months.

The activity of eNOS, in the presence or absence of eNOS co-factors, did not differ between the two mouse strains suggesting that the difference in bioavailable NO attributable to selective IL-1 inhibition is mediated by increased consumption of NO via increased arterial oxidative stress.

The phenotype of these two mouse strains suggests that increased IGF1 activity in tissues leads to uncontrolled growth.

In the present study, we observed statistically and physiologically significant differences in vascular development and hemodynamics between two mouse strains, suggesting a prominent role for genetics.

Percentages of infected PMNs were similar between mouse strains, suggesting that the absence of IFNAR1 did not affect bacterial uptake by these cells.