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Following irradiation of mouse skin with UVB, the level of phosphorylated Stat3 was initially decreased and then increased significantly at later time points in epidermis (Fig. 7C).
Clearly, pretreatment of mouse skin with solid microneedles significantly increased the permeability of 5-FU through the skin.
Pretreatment of mouse skin with various amounts of baicalein caused inhibition of H2O2 and myeloperoxidase formation by 12-O-tetradecanoylphorbol-13-acetate 12-O-tetradecanoylphorbol-13-acetate 12-O-tetradecanoylphorbol-13-acetate 12-O-tetradecanoylphorbol-13-acetate 12-O-tetradecanoylphorbol-13-acetate
Data in Fig. 1 clearly demonstrated that pretreatment of mouse skin with microneedles significantly increased the permeability of 5-FU through the skin.
Storm et al. (1990) compared CYP-dependent activities in the mouse skin with those in the skin of the guinea pig and human.
Topical exposure to oxazolone induced a delayed-type inflammation in the mouse skin, with increased infiltration of inflammatory cells (Fig. 2a).
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We observed a larger number of neutrophils in the skin of mice infested with ticks treated by Ir-LBPsiRNA than in mouse skin infested with either untreated ticks or ticks treated with irrelevant siRNA or PBS.
It was recently shown that the capacity for excision repair in mouse skin oscillates with a circadian rhythm that is correlated with rhythmic XPA protein oscillation.
Mouse skin fragments with 5-day engorged ticks were embedded in TissueTek (Sakura) and stored at −20°C.
The objective of this investigation was to characterize, the transcriptional response and the onset of regenerative processes in mouse skin irradiated with different doses of fast neutrons.
In a recent study, persistence of damaged DNA has been demonstrated in mouse skin irradiated with high doses of X rays.
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