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The SVCT2 knockout mouse shows a severe phenotype with lung failure and brain hemorrhage leading to early postnatal death [38].
The A53T transgenic mouse shows a significant [P<0.05, n = 3] decrease of 30.7% in TH positive DA neurons of the Substantia nigra versus wild type.
The Ts1Cje mouse shows a recognizable DS phenotype while still allowing for breeding, and was consequently chosen as a model in this study.
Only the presumed immigrant mouse shows a different assignment and associates with populations from mainland Germany.
The CTLA-4 knockout mouse shows a dramatic phenotype [ 2, 54].
Pdgfrb has previously been shown to be expressed in mesangial cells, and the Pdfrb mutant mouse shows a failure of mesangial cell development [ 34].
Similar(47)
Of note, cochlear tissues of Cmah-null mice showed extensive apoptosis, whereas control mouse showed a moderate apoptosis.
The mouse showed an opposite orientation of the region internal to the palindrome.
Ets1 deficient mice show a substantial reduction of ILC2s in the bone marrow and lymph nodes.
GnT-IVa-deficient mice showed a metabolic disorder subsequently diagnosed as type 2 diabetes.
Twenty-four hours after lipopolysaccharide administration mice showed a reduction in the total number of nose pokes.
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