Sentence examples for mouse should provide from inspiring English sources

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The Id2−/− mouse should provide unique opportunities to study the progression of neurodegenerative disorders involving the dopamine system.

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Future investigations employing genomics/proteomics and novel genetically altered mice should provide further insight into how n-3 PUFAs modulate these diseases as well help to identify clinically relevant biomarkers.

The factors that initiate lesion formation are unknown, but disruption of these genes in mice should provide animal models to address these and other important questions about the pathogenesis of HHT.

Together, these efforts to genotype and phenotype mutant mice should provide additional data that could aid in validating candidate disease genes identified in GWAS.

Propagation and interrogation of tumours from these clones in iNOS knockout mice should provide an even more unambiguous assessment of the contribution of tumour cell-derived iNOS.

Thus, investigation of BACE1 elevation in 5XFAD mice should provide valuable insight into the formation and progression of amyloid plaques in AD.

However, considering that metabolic syndrome is a combined disorder involving both the hypothalamus and the pancreas, the analyses of PHT- Atf3-KO mice should provide more practical insights into the relationship between ATF3 and the metabolic syndrome.

"These mouse models should provide a major boost to research efforts to develop new treatments for the common cold, as well as for more potentially fatal illnesses such as acute attacks of asthma and of chronic obstructive pulmonary disease," he said.

Given the fact that similar changes are observed in 99mTc-MAG3 renogram in human patients with renal obstruction [ 26, 27], combination of this imaging technique with mouse UUO models should provide an effective assay for evaluating treatment protocols of this disease or screening new pharmacological agents as well as for elucidating pathophysiological mechanisms of obstructive kidney disease.

Profiles of behavioral changes in other mouse models of ASD should provide additional specificity in the search for biomarkers associated with particular ASD symptoms and symptom clusters.

While exact replication of AD in mice may not attainable (due to phylogenetic differences and fundamental differences in behavioral ecology), rigorous comparative analysis of cognitive behavior observed in various mouse models of AD should provide a framework for better understanding of molecular mechanisms underlying cognitive impairment observed in AD patients.

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