The same methodology was used for human and mouse samples, including electrophysiological recordings, biocytin filling, histological processing, and reconstruction methods.
The comparison between corresponding promoters in human and mouse samples, including the novel promoter p1@FOXG1 in human and pA@Foxg1 in mouse, revealed remarkably similar shapes, suggesting evolutionary conservation in their regulation.
Here we report CNV analysis of publicly available, high-density microarray data files for 351 mouse tail samples, including 290 mice that had not been characterized for CNVs previously.
Table 1 lists the phenolic compounds measured in the mouse plasma samples, including their concentrations in animals dosed by oral gavage and by sc injection.
The analyzed data represent a comprehensive collection of most experiments (9 in total) that have evaluated the effect of aging in the laboratory mouse or human samples, including the cardiac-specific samples generated as part of the AGEMAP project [ 88] (see Additional file 3).
We also observed that SALL4 was able to bind the promoter regions of these two genes in either mouse ESCs, two human AML samples including M0 (FAB classification) or AML transformed from CML (chronic myeloid leukemia) using ChIP-on-ChIP assays (data not shown).
In this case, the mouse model offers several advantages over the human samples, including samples being harvested from mice with identical genetic backgrounds, as well as whole spine samples being obtained for global gene expression profiles.
The original spectra were obtained from LC/MS data of some biological samples including human cells, mouse tissues, and plant species.
A total of 69 samples were tested for F. tularensis by culture and direct PCR: 49 were spleen samples, including samples from mice not selected for pathologic investigation because of severe autolysis, and 20 were abdominal swab samples, which were used when the extreme grade of autolysis prevented the unambiguous identification or collection of the spleen following dissection (5 ).
The MDGA has been used to characterize and map the subspecific origin (from the three main Mus musculus subspecies; domesticus, musculus and castaneus) and haplotype diversity of SNPs for 198 samples including wild-caught mice, wild-derived laboratory strains and classical laboratory strains [ 23].
Then, 39 blind samples, including 27 tissues of mice infected with Y. pestis and 12 tissues of healthy mice as negative control, were detected with the FOB-3.
