Sentence examples for mouse retina we from inspiring English sources

To determine the localization of NR1D1 protein in the mouse retina, we first performed Western blotting to confirm antibody specificity.

To examine the SPIG1 mRNA expression in the developing mouse retina, we performed in situ hybridization on coronal sections at P5 (Figure 1A).

After 4-day culture of adult mouse retina, we were able to observe EGFP expression in morphologically intact retinal ganglion cells (Figure 5D), more than 50 to 100 cells per retina.

To determine whether the gene relationships represented in the fly seed network are represented in the developing mouse retina, we first converted the literature-based fly seed network into a mouse gene network of putative homologs (Table 1).

In an attempt to identify the HCN channel isoforms contributing to Ih in RBCs of the mouse retina, we examined this issue in detail by immunohistochemistry, using commercially available isoform specific polyclonal antibodies.

Although we failed to detect Nkx2.2 in the mouse retina, we found widespread expression of Sox2 and Sox9; these factors were present in the nuclei of Müller glia (Figs. 2a and 2e), consistent with previous reports [12], [13].

By flow-sorting immature Crx-expressing cells from various developmental time points and transplanting the genetically labelled populations into wild-type or degenerating mouse retinae, we were able to compare their integration competency and ability to generate cones after transplantation.

In addition, by analyzing the changes of miRNA expression levels during the physiological process of light adaptation (LA) to dark adaptation (DA) of mouse retinas, we observed a negative correlation between miRNA stability and miRNA level, although the correlation is not significant (R = −0.14, P = 0.08) but shows an obvious tendency.

HGSNAT is highly expressed in the mouse retina, and we hypothesize that the retina requires higher HGSNAT activity to maintain proper function, compared with other tissues associated with MPS IIIC, such as the brain.

For the case of the mouse retina data, we consider maximum link probability p m a x ∈ { 0.95, 0.9, 0.7 }, variance scales for the synapse density profile of σ 2 ∈ { 0.01, 0.1, 1.0 } (of normalized depth), and K ∈ { 2, 3 } possible synapse density profile mixture components.

In mouse retina with EIU, we demonstrated that a lack of iNOS attenuated leukocyte rolling along the major retinal veins and subsequent leukocyte migration into the retina.