Exact(1)
When implanted orthotopically in the mouse prostate, no significant differences were observed in tumor growth between both groups (Fig. 3a).
Similar(59)
Morey Kinney et al. (2009) demonstrated that administration of 0.3% green tea polyphenols (GTPs) to wild-type (WT) and transgenic adenocarcinoma of mouse prostate (TRAMP) mice showed no alteration in 5-methyl-deoxycytidine (5mdC) levels in prostate, gut, and liver from WT mice at both 12 and 24 weeks of age, with the single exception of a decrease of 5mdC in the liver at 12 weeks.
Spop localized in nuclear foci similar to human LNCaP cells in mouse prostate cells (MPCs) after IR, with no alteration in foci by expression of SPOP-F133V.
Furthermore, we found G-1 to have little or no effects (weight and histology) on mouse prostate and on growth-quiescent immortalized benign prostatic epithelial cells in cultures.
In addition, no obvious TgTSPY-positive cells were detected in the TgTSPY9/CD1 mouse prostate at 14 weeks of age.
Transgenic adenocarcinoma of the mouse prostate.
We used animals designed to develop this disease, Transgenic Adenocarcinoma of the Mouse Prostate (TRAMP) mice, to investigate the potential role of AHR signaling in prostate cancer development.
Primary prostate colonies from DsRED transgenic mouse prostate cells were trypsinized and dissociated into single cells.
Study of tumor development in mouse prostate cancer models can be instrumental in understanding human prostate cancer.
RNA from microdissected mouse prostate stroma and epithelium, and human prostate fibroblast (PSC27) were used as template for qRT-PCR.
Moreover, overexpression of Skp2 in mouse prostate leads to prostate intraepithelial neoplasia (PIN) [50].
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