Sentence examples for mouse myoblasts produced from inspiring English sources

linc-MD1 RNAi-dependent downregulation in mouse myoblasts produced a decrease in the accumulation of myogenic markers, while its overexpression led to increased synthesis.

Although skeletal myoblasts produce several factors [17], the majority of these are positive regulators of angiogenesis.

In our latest experiment we targeted the last exons of the nebulin transcript produced by C2C12 mouse myoblasts.

Targeted exon exchange requires the spliceosome provided by the cell in addition to the target pre-mRNA and pre-trans-splicing molecules (PTMs) both produced from expression vectors transfected into C2C12 mouse myoblasts.

The effect of MstnPP soluble aggregates, protofibrils and fibrils on the viability of C2C12 mouse myoblasts was investigated by monitoring the absorbance of formazan (Fig. 8) produced after addition of WST-1 (Roche).

Methods: Mouse myoblasts (C2C12 cell line) were cultured in the presence of type 1 or type 2 IFNs and ISG15 expression assessed by microarray analysis.

Mouse myoblasts (C2C12), human embryo kidney cells (HEK293), and human umbilical vein endothelial cell lines (ECV304 and EA.hy926) were cultured with high glucose DMEM.

We choose primary mouse myoblasts as a second test bed because of their importance in treating genetic muscular dystrophies, including Duchenne muscular dystrophy.

We identified and quantified (i) metabolic differences between genetically engineered human cell lines, (ii) alterations in cellular metabolism induced by differentiation of mouse myoblasts into myotubes, and (iii) metabolic changes caused by activation of neurotransmitter receptors in mouse myoblasts.

We further seek to identify the expression kinetics of various periostin isoforms during the differentiation of rat and mouse myoblasts.

Upper panel: Time-sequence images showing the process of segregation in mixed co-cultures of C2C12 mouse myoblasts (red) and HaCaT human keratocytes (green).