Sentence examples for mouse models show from inspiring English sources

Both mouse models show that TLX1 can induce T-ALL.

Since all circadian gene-mutant mouse models show increased sensitivity to γ-radiation, we conclude that the molecular clock functions in tumor suppression in vivo.

However, current humanized mouse models show sub-optimal human T cell reconstitution and limited ability to support immunoglobulin class switching by human B cells.

Astrocytes from DS fetal brain tissue and DS mouse models show increased concentration of intracellular calcium [55], [56], altered sensitivity to oxidative stress [57], deficits in mitochondrial energy metabolism [22], [58] and abnormal APP transport and secretion [22].

We observed a reduction in HPC proliferation in 3-month-old APPPS1 mice but not in R1.40 animals (Fig. S2) even though both mouse models show elevated levels of Aβ.

These compensatory mechanisms, which are most likely controlled by a negative feedback system that keeps overall neuronal activity at physiological levels, could explain why some mouse models show remarkably few functional impairments and symptoms even with large plaque loads in the cortex.

The combination of SS1P with chemotherapy is based on results from in vivo mouse models showing marked synergy between SS1P and chemotherapy [23] [26].

OA cartilage in both humans and mouse models shows increased expression of HIF-2α.

Three mouse models showed deficits in the response probability during training.

Two different mouse models showed that Foxf1−/− embryos die by 8.5 days postcoitum (dpc) from defects in extraembryonic mesoderm development.

These are the first mouse models showing that IGF-IR or IRS overexpression leads to tumorigenesis in vivo.