Sentence examples for mouse models leads from inspiring English sources
These results show that Rac1 depletion in both mouse models leads to the loss of thymic epithelial cells and destruction of the normal thymic architecture.
Administration of Type I IFN in mouse models leads to reduced skin reactivity to lipopolysaccharides (LPS), reduced response to ischemic insult/chemokine injection, and protection from collagen-induced arthritis (CIA), experimental autoimmune encephalomyelitis (EAE) and delayed-type hypersensitivity-allotransplant rejection (reviewed in [3]).
Despite these multifactorial control mechanisms, mere overexpression of Yap in the whole liver in mouse models leads to hyperplasia, organomegaly, dedifferentiation, and cancer (Camargo et al., 2007; Dong et al., 2007; Schlegelmilch et al., 2011), raising a question as to whether some layer of homeostatic growth control may be lacking in such models.
Both mouse models led to significant pulmonary vascular remodeling consistent with our prior demonstration of structural and hemodynamic PH.
Both R-IVET screenings using the bacteremia and peritonitis mouse models led to the identification of 64 putative genes that are activated during the pathogenic process.
The data from the different mouse models led to similar observations.
In the study, the results showed that two mouse models led to significant PHT and PHG, upregulated gastric mucosal PUMA expression and induced mucosal epithelial apoptosis.
The exciting results obtained in mouse models led to a clinical trial to investigate the safety of mononuclear cell injection in patients with CCC.
Lee et al. showed that injection (i.p). of LPS into mouse models led to increased levels of Aβ and Tau aggregation [ 242].
The finding of an overall increase in the extractability of matrilin-3 and COMP by buffer 1 in all three PSACH-MED mouse models led us to consider that some of this protein could represent intracellular retained material.
Nanoparticle delivery of miR-506 in orthotopic ovarian cancer mouse models led to E-cadherin induction and reduced tumor growth, whereas delivery of miR-200 members into the tumor endothelium resulted in marked reductions in metastasis and angiogenesis and induced vascular normalization.
