Sentence examples for mouse models lacks from inspiring English sources

The coexpression of RET/PTC 3 and E7 in transgenic mouse models lacks any cooperative effect in the neoplastic transformation of thyroid cells and the E7-induced thyroid phenotype is dominant with respect to the RET/PTC3 one.

Indeed, they have proved to be essential elements in normal physiology, as shown by mouse models lacking these proteins, that evidence several developmental abnormalities and pathological features.

Mouse models lacking the α3- or γ2-chain of laminin-332 have been developed and a spontaneous ß3-knockout mouse exists, but all die shortly after birth.

We therefore used two different conditional mouse models lacking Prkg1 in the nervous system.

Further tests in mouse models lacking the specific epithelial receptor will permit more targeted examination of the complex interplays between the pathogen and the lung epithelium.

To characterize the influence of PRKG1 on sleep and circadian rhythmicity in mammals, we analyzed two different mouse models lacking functional PRKG1 in the brain.

The complexity of the dual roles played by both estrogens and androgens is illustrated by studies in mouse models lacking either the androgen receptor (ARKO) or the estrogen receptor alpha (ERαKO).

Support for these activity-dependent roles of AChR derives from in vivo studies of mouse models lacking synaptic organizers such as agrin or MuSK, showing apparently normal myofiber growth and survival [4], [7].

We demonstrate that Rb does in fact play an active role in control of CM cell cycle progression, and in contrast to mouse models lacking Rb in heart, acute loss of Rb appears to be a central regulator of the G1/S transition in developmentally early stage CM, consistent with most reports on Rb in other non-cardiac systems.

Mouse models lacking these components will be a invaluable resource to test this hypothesis.

Mouse models lacking expression of Cx26 or Cx30 confirmed that these proteins are essential for hearing.