Sentence examples for mouse model supplementary from inspiring English sources

We first checked the effect of pazopanib in vivo tumor bearing mouse model (Supplementary Fig. 7).

These results suggest that HMGB1 does not trigger brain inflammation in our mouse model (Supplementary Fig S1).

This motoneuronal expression pattern was also preserved in an ALS patient (Fig. 2D F) and the SOD1(G93A) mouse model (Supplementary Fig. S1).

To study the role of Ankrd2, we took advantage of the Ankrd2 knockout (KO) mouse model (Supplementary Figure S1) and performed global transcriptome analysis on proliferating, fusing, and differentiated primary myoblasts derived from wild-type (WT and KOO mice infected with adenovirus expressing HA-tagged Ankrd2 (AdAnkrd2) or control GFP (AdGFP).

These findings stimulated many studies in Lambert Eaton myasthenic syndrome; Peter Molenaar showed that acetylcholine contents of Lambert Eaton myasthenic syndrome muscle were normal, but confirmed that the release was deficient (Supplementary material B42), as also found in the mouse model (Supplementary material B60).

Importantly, 14 out of these 169 predicted piRNA target genes have been shown to be essential for spermatogenesis in mouse models (Supplementary information, Table S3).

Hematoxylin and eosin staining of tumour sections clearly showed PanINs as well as areas with well-developed tumours compared with the control pancreas from contemporary littermates of both KC: K-rasG12D; Pdx1-Cre (40th and 50th weeks) and KPC: K-rasG12D; Trp53R172H/+; Pdx1-Cre (5th, 10th, 15th, 20th and 25th weeks) mouse models (Supplementary Figures 1a and b).

It was interesting to note a similar increase in hepatic p-CREB in P2 Smn +/−;SMN2 and P21 Smn 2B/− mice, an intermediate SMA mouse model (see Supplementary Material, Fig. S1).

To investigate the involvement of this pathway in the increase of [Ca2+]i we deleted Gαq and Gα11 proteins in glial cells and tanycytes with the help of the Cre-driver mouse line GlastCreER T2 (Gα q/11 gliaKO mice; for an overview about mouse models, see Supplementary Table 1).

In this study, we first showed that HMGB1 supplementation is effective in an SCA1 mouse model (Fig 1, Supplementary Figs S1 and S2) and that the therapeutic effect by AAV-HMGB1 was expected even after the onset of symptoms (Fig 8, Supplementary Fig S8).

To determine whether TECK is involved in Treg differentiation and the development of endometriosis in vivo, we used the C57B/L6 mouse i.p. endometriosis model (Supplementary Figure 5A).