Sentence examples for mouse model designated from inspiring English sources

This mouse model, designated "human only" (HO), exhibits severe elevations in both plasma and tissue levels of Hcy, methionine, S-adenosylmethionine, and S-adenosylhomocysteine and a concomitant decrease in plasma and hepatic levels of cysteine.

We now report the development and characterisation of a new FRDA mouse model, designated YG8sR, derived from natural breeding of YG8R mice.

We now describe the cellular, molecular and behavioural characterisation of a newly developed YAC transgenic FRDA mouse model, designated YG8sR, which we have shown by DNA sequencing to contain a single pure GAA repeat expansion.

To test the possibility that truncated BRCA1 proteins might have a dominant effect on wild-type BRCA1 or other proteins, we generated a targeted mouse model, designated Brca11700T, in which a neomycin resistance gene is inserted in exon 20 of the murine Brca1 gene.

We have utilized two previously published mouse models for this study: 1) the pro-arthritic mouse model (designated SKG) injected with metastatic BC cell line (4T1) in the mammary fat pad, and 2) the PyV MT mice that develop spontaneous mammary gland tumors injected with type II collagen to induce autoimmune arthritis.

The Bf 109 fighters were a navalized version of the "E" model, designated as Bf 109T.

In the current study, we focused on the effects of bacterial endotoxin in vitro on proliferation of A549 cells, a cell line derived from human lung adenocarcinoma, in an ex vivo short-term cultivation model designated short-term stimulation of tissues (STST) using human specimens obtained from patients with NSCLC and in vivo in the subcutaneous A549 adenocarcinoma mouse model.

Herein we report the characterization of a novel mouse model of NPH (designated p23-ST1), created by N-ethyl-N-nitrosourea (ENU -induced mutagENU -induced

Results Here, the authors report on the development and characterisation of a novel FRDA mouse model from YG8R breeding, designated YG8sR.

Our study suggests that loss of TIMP3 is a hallmark of DKD in human and mouse models and designates TIMP3 as a new possible therapeutic target for diabetic nephropathy.

These 4 models included an A-DROP model and 3 newly developed models (designated Models 0, 1, and 2).