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More studies, such as miRNA injection in ARDS mouse model, are needed to proof their functionality.
The natural history and the mechanisms behind the alteration of vaginal distension (VD) in a mouse model are not clear.
In 2013, Kosaka et al. revealed that knockdown of nSMase2, which is required for the synthesis of ceramide, EV secretion and miR-210-3p miR-210-3p miR-210-3pd, and angiogenesis and metransfer is a xenograft mouse model are suppressed [25].
Nevertheless, specific muscle studies in this knockout mouse model are still required to exclude a muscle associated phenotype.
Similar to human TLX1-positive T-ALL, the T-ALL cells in our mouse model are arrested at the double-positive stage of differentiation.
This is consistent with our observation that the growth and biochemical properties of Grp94+/+ and +/− ESCs and MEFs isolated from our Grp94 mouse model are indistinguishable (data not shown).
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A subcutaneous xenograft nude mouse model was established.
The immunosuppressed mouse model was induced by cyclophosphamide.
An encephalitis mouse model was established via intracranial HV administration.
Liver-injured mouse model was established by CCl4 injection.
A nude mouse model was utilized to demonstrate the osteoinductive potential of the scaffold in vivo.
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