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The new mouse model also gives researchers a new way to test potential autism drugs before trying them in human patients.
The in vivo mouse model also demonstrated that the chemical inhibition of Nrf2 can increase cisplatin (CDDP) cytotoxicity.
Our xenograft mouse model also showed that ursolic acid combined with oxaliplatin achieved the best tumor control effects among the strategies tested.
The mouse model also enabled the testing of new drugs against progressive MS. In the study, nanoparticles creating tolerance to the myelin antigen were administered and prevented progressive MS from developing.
Using the APP-PS1-21 model model, also specific binding of [ 18 F]florbetapir was shown to increase from age 3 to 8 months; however, no further increase was detected at 12 months compared to the baseline scan at 3 months [11].
The myostatin-null mouse model also provides insights into the physiological role of this protein.
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In mouse models also IL-27 had a strong anti-tumor effect, which was enhanced by the presence of IL-12 [34], [35], [36], [36].
Gene knockout mouse models also support this idea.
Diabetic mouse models also display an increase in myocardial oxygen consumption.
On the other hand, mouse models also have their limitiations (Box 2).
Cells isolated from seipin knockout mouse models also exhibit impaired adipogenesis.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com