Sentence examples for mouse lung after from inspiring English sources

Sox9 protein expression level is high in embryonic lungs but drastically decreases in mouse lung after birth.

In this study, we confirmed several significant alterations in gene expression by transcriptome profiling in the mouse lung after chronic low-dose (10 and 100 ppb) As exposure.

To ensure that the level of MIR2911 in mouse lungs is high enough to play a biological role, we calculated the amount of MIR2911 in mouse lung after gavage feeding with HS decoction.

Gestational arsenic exposure increased expression of the estrogen receptor-α gene (ER-α) and genes related to estrogen signaling in the female fetal mouse lung after environmentally relevant exposures via drinking water (Shen et al. 2007).

Blood vessel growth was hyperstimulated in the hyperoxic mouse lung after D5 such that by D28 the earlier deficit had disappeared and both the number of blood vessels per section area and the Pecam1 levels had reached control levels.

So neonatalogist Jeffrey Whitsett at Cincinnati Children's Hospital Medical Center, Ohio, and colleagues devised a way to turn the Foxa2 gene off in mouse lungs after the fetal mice had mostly developed but before their lungs fully matured.

It has been shown that TGF-β is an important mediator for the development of BPD [79 81] and is activated in neonatal rat lungs after hyperoxia exposure [82] as well as in neonatal mouse lungs after mechanical ventilation with mild hyperoxia [83].

One H37Rv strain (termed JHU, obtained from the Johns Hopkins Hospital clinical microbiology laboratory [14]) had been passaged multiple times through mouse lungs after intravenous infection.

Our goal was to develop an approach for measuring the relative abundance of individual mutants in mutant mixtures that can be applied to any kind of mutant, is highly sensitive, has a large dynamic range and allows to accurately measure growth and survival of Mtb mutants in mouse lungs after aerosol infection.

One study (Rivera et al. 2002) demonstrated inducibility of CYP2S1 in mouse lungs after systemic administration of dioxins.

The level of MIR2911 in mouse lungs after drinking HS decoction increased by > 8-fold, whereas no change in the MIR2911 level was observed in control mouse lungs.