Sentence examples for mouse lines revealed from inspiring English sources
The analysis of a battery of Cre transgenic mouse lines revealed that SVZ restricted NSCs derive from both cortical RG cells and cells of lateral Ganglionic Eminence [15], [16].
Interrogation of our ongoing analysis of impulsivity in BXD recombinant inbred mouse lines revealed an association of impulsive responding with levels of alpha-synuclein expression in hippocampus.
However, crosses into the Rosa26LacZ and mT/mG reporter mouse lines reveal robust and widespread Cre activity in neuronal tissues at E15.5 and E10.5 that is not strictly oligodendrocyte lineage specific.
Comparison of both mouse lines reveals a stronger reactivity of SVZdNP than pOPC during the early phases (i.e. during demyelination) since between W3 and W5 YFP+ cell density is multiplied by 4 in NestinCreERT2-YFP mice and only by 2.5 in PDGFRaCreERT2-YFP mice.
Histological evaluation of the miR-9-Sp/tdTomato reporter mouse line revealed a strong signal for both proteins in most organs, indicating the absence of miR-9 activity.
Last but not least, results from a third DUSP4-deficient mouse line revealed phenotypes in CD4 T cell proliferation that could be attributed to altered IL-2 response [ 59].
Recently established AM knockout mice lines revealed that AM is essential for development of vitelline vessels of embryo.
Analysis of this miR-9-SpNestin-Cre mouse line reveals a prominent role for miR-9 on postmitotic neuronal differentiation as a regulator of dendritic development through repression of REST expression.
In vitro screening for mouse tumor cell lines revealed that all mouse lines we tested exhibited comparably low susceptibility to infection by our attenuated mutant virus, on par with normal quiescent human cells.
Cell-free transcription systems of mouse-human hybrid cell lines revealed that the promoter for mouse or the promoter for human rRNA would not function in a cell-free extract made from another species [116] [119].
Analyses of several mutant mouse lines have revealed that transcription factors, guidance cues, adhesion molecules and guidepost cells control the navigation of thalamocortical axons at specific decision points within their intermediate targets [ 1- 5].
