Sentence examples for mouse lines on from inspiring English sources

Heterotopia were observed in numerous genetically-engineered mouse lines on a congenic C57BL/6 background.

We maintain our mouse lines on a 129X1/SvJ C57BL/6J mixed background.

Similar results were obtained using both of these VGF mutant mouse lines on C57Bl6 backgrounds.

The raw sequencing read files (FASTQ) for the 8 mutant mouse lines on which the results of this article are based have been made available at the ENA, with study accession number PRJEB8962 at http://www.ebi.ac.uk/ena/data/view/PRJEB8962.ac.uk/ena/data/view/PRJEB8962

In this study, we investigate the contribution of allelic differences to phenotypic variability and to COL4A1 and COL4A2 biosynthesis using a series of Col4a1 and Col4a2 mutant mouse lines on a uniform C57BL/6J genetic background.

To gain insight into the molecular and cellular changes that may be responsible for the increase in energy expenditure and decrease in adipose stores detected in VGF knockout mice, we examined the effect that targeted ablation of VGF has on the regulation of gene expression in adipose tissues, analyzing two independent VGF knockout mouse lines on C57Bl6 backgrounds.

In some experiments, 12-week old male LysEGFP mice, a genetically engineered mouse line on a C57BL/6 background possessing green-fluorescent myeloid cells (mostly neutrophils) as a consequence of 'knockin' of enhanced green fluorescence protein (EGFP) into the lysozyme M gene, were used [47], [48].

We studied another TGFα-expressing mouse line on a CD1 background, whose gastric mucosa appears normal.

Cngb3−/− mouse line (on C57BL/6N background) was generated by targeting deletion (Deltagen Inc., San Mateo, CA) as described previously (31).

Transgenic mice were further back-crossed for 6 generation to obtain a mouse line on a pure C57Bl/6 J genetic background.

The mutant mouse lines originated on a mixed C57BL/6 and DBA/2 background and were backcrossed on to the C57BL/6J background; APP expression is directed to neurons under the control of a human platelet-derived growth factor β polypeptide promoter.