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The (μ gt, ϕ g t )'s were sampled as pairs from the mouse limb case study data described in the next section.
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Similar results are demonstrated in a case study of the adult mouse limb presented in Section 3.2.
Here, we analyze the emergence of different MSC types during mouse limb and long bone development.
Tumor cells (5 × 105 in the volume of 100 μl) were transplanted intramuscularly into the mouse limb.
Therefore, Shh specifically becomes necessary for normal limb development at or just distal to the stylopod/zeugopod junction (elbow/knee joints) during mouse limb development.
The Hox genes have recently been shown to regulate Shh transcription in developing mouse limb buds.
This indicates VBP15 increases functional mouse limb strength.
(G ) Comparison between S. canicula fin and mouse limb.
Their name originates from the mouse limb deformity gene.
Thus, mouse limb ischemia was induced by ligation of the femoral artery and its branches in the left hind-limb of SCID mice.
The HOXA/ Hoxa cluster is implicated in regulating mouse limb bud development (especially Hoxa9– Hoxa13) [ 14].
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