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RNA used as positive control was from mouse kidney, mouse brain (Ambion, Austin, TX).
We supplemented our analysis with control animals from three previous experiments that had been conducted by our lab [ 35, 37, 46] to expand our coverage of species and tissue types across mouse kidney, mouse liver, rat adipose, rat hypothalamus and rat liver.
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In the new paper, Dr. Mendelsohn and others showed in the mouse embryo and cultured embryonic mouse kidney that depriving the fetus of vitamin A could drastically reduce the amount of ret in the developing kidney tissues and also disrupt formation of kidney structures.
Unlike human kidney development, mouse kidney development continues postnatally.
A conceptual developmental framework has emerged for the mouse kidney.
Park, J. et al. Single-cell transcriptomics of the mouse kidney reveals potential cellular targets of kidney disease.
Mouse kidney progenitor cell.
We compared 25(OH D-1 alpha-hydrOH D-1e activity in the Hyp-mouse kidney to that in normal and phosphactivityeted mouse kinney.
Park et al. Single-cell transcriptomics of the mouse kidney reveals potential cellular targets of kidney disease.
Renin gene expression in the mouse kidney and submandibular gland (SMG) are differentially regulated by cAMP.
Altered intragraft immune responses and improved renal function in MHC class II-deficient mouse kidney allografts.
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