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Our data derived from biodistribution experiments in the Rip1Tag2 mouse indicate that at least in the presented experimental model, there is no such window.
It remains unclear when the human fetal reno-hepatic axis for endogenous Cr synthesis is established, although studies in the precocial spiny mouse indicate that this axis develops late in gestation [64].
Recent studies in the mouse indicate that IL18 has a role in promoting an IFN-γ dependent positive regulation of memory CD8 T cell proliferation [44].
Studies in the mouse indicate that αβ-T cells are not required during the first week of Salmonella infection [69], [70] and IFNγ is produced in response to IL12 and IL18 in rag−/− and SCID mice that lack T cells [71].
Recent studies of the GBP5 knockout mouse indicate that GBP5 functions in host defense, inflammasome assembly, and inflammatory response [ 7].
Several evidences obtained in the mouse indicate that long ncRNAs control genomic imprinting by distinct mechanisms in embryonic and extra-embryonic tissues.
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Anoikis was suppressed in the non-adherent culture of hepatocytes isolated from gld/gld mouse, indicating that Fas signal induces hepatocyte anoikis.
Recent studies using conditional knockout mice indicate that Notch1 as well as Notch2 plays an important role postnatally in liver development and in maintenance of hepatic function.
Transepithelial resistance was not altered in Tg and Ko mice, indicating that tight junctions were not affected.
A similar distribution was observed in K5cre-CMVcaNrf2 mindicatingathat tighttight junction permeability is not grossly affected (Fig 5B).
A study performed in mouse indicates that retinoblastoma arise from a precursor cell that is naturally death-resistant [34].
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