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Previous work by our group has shown that in the Mg-depleted mouse hypomagnesemia develops and skeletal Mg content falls significantly [ 10].
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"When healthy mice were co-housed with mice that had altered gut microbes, the healthy mice also developed a susceptibility for development of liver disease and obesity".
The ApcΔ716 mouse develops ∼300 polyps, Apc1638N mouse develops ∼3, ApcΔ14 mouse develops ∼65, Apc mouse develops ∼35, ApcΔ580 mouse develops ∼120, and ApcΔ474 mouse develops ∼30 [16].
The fascin-deficient mouse develops largely normally.
A mouse is injected with the target protein causing the mouse to develop thousands of antibodies to it.
Both transgenic mouse lines develop fatal BPL.
Two transplanted mice out of twelve mice developed T-ALL.
Renal function tests showed that SPAK234A/234A mice actually developed a Gitelman-like syndrome with hypomagnesemia and hypocalciuria.
She reports that when this RNA is injected into the eggs of normal mice, the eggs develop into mice with the same white extremities.
Analysis of these mice showed that Hif2α−/− mice developed pancytopenia.
On the face of it, the mouse developed normally.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com