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Oligomeric A β injection into the WT mouse hippocampus induced neuronal cell death in 20 days after injection.
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These findings suggested that an intrinsic apoptosis pathway in the mouse hippocampus was induced by TiO2 NP exposure, which resulted in impaired spatial recognition ability [62].
This possibility is also supported by data showing that activation of PKC with phorbol ester in mouse hippocampus tissues induced a rapid demethylation of the reelin promoter [ 56].
Furthermore, CP treatment decreases neuronal cell death in the hippocampus induced by chronic BCCAo.
Moreover, KA-induced seizures increase endocannabinoid levels in the normal mice hippocampus, and induce more severe neuronal damage in the hippocampus of CB1 knockout mice [101].
The formation of model Hirano bodies in the mouse hippocampus did not induce neuronal loss or impact long-term plasticity.
Moreover, Rib induced astrocyte activation in mouse hippocampus and impaired spatial learning and memory ability.
Moreover, injection of Rib induced astrocyte activation in mouse hippocampus and impaired spatial learning and memory abilities.
Regardless, these data are consistent with Bmf fulfilling an apical role in the signaling pathways induced by SE in the mouse hippocampus.
Therefore, current study was designed to investigate the effect of quercetin on chronic unpredicted stress (CUS) induced behavioral dysfunction, oxidative stress and neuroinflammation in the mouse hippocampus.
Our study newly suggests that H4K5ac is induced in an activity-dependent manner in the adult mouse hippocampus where it may prime genes for rapid expression following repetitive learning.
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