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The formation of model Hirano bodies in the mouse hippocampus did not induce neuronal loss or impact long-term plasticity.
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Only in the CA3 region of the hippocampus did the mouse lines display no differences.
Scrapper transgenic mice overexpressing SCRAPPER in the hippocampus did not show any significant difference in every test argued in this manuscript by comparison with wild-type mice.
However, an i.c.v injection of Res or NE into the hippocampus did reverse age-related LTP impairments in SAMP8 mice (Fig. 5F H).
Yet the caudate and hippocampus did.
If the hippocampus does produce new cells, are there enough to be any significance?
The hippocampus does so, encoding the firing sequence in the cortex and thereby consolidating the memory.
This shows that the overlap between our probes is highly significant, and that these 2971 genes really do coexpress with Mgst3 in the mouse hippocampus.
In order to do so, we performed time-course MALDI imaging of mouse hippocampus on both control and kainate-administered mice at 30 and 180 minutes after administration.
Consistent with this report, we found that deleting α5 did not change the potency of nicotine at triggering [H]-NA release in the mouse hippocampus, suggesting that the nACh receptors that modulate NA release system differ between the hippocampus and the Hb-IPN system.
When tested in mice, DIM attenuated LPS-induced brain inflammation in mouse hippocampus.
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