Sentence examples for mouse heart to from inspiring English sources

Previous studies in our lab, demonstrated the ability of the mouse heart to compensate functionally for significant structural damage after previous irradiation [ 17].

For example, a study in 2014 even demonstrated the capability of CSCs isolated from mouse hearts to differentiate into sinus node like cells when co-cultured with mouse sinus node tissue [75].

Now, researchers reveal that they have nudged cells within mouse hearts to repair some of the damage, a discovery that might prompt new treatments for heart attacks in humans.

Several studies reported an upregulation of CTGF upon ischemic conditions.[23], [24] In order to examine the effects of CTGF overexpression on the ischemic heart disease, we subjected isolated CTGF-TG and WT mouse hearts to 40 minutes ischemia with subsequent 60 minutes reperfusion phase.

To model this situation, I evaluated the efficacy of using H3K4me1 and H3K27ac collected in mouse hearts to predict known human heart enhancers.

The characteristic interstitial fibrotic response in the compensating myocardium remote to the infarct also did not occur in hyperglycemic EphA2-R-M mouse hearts to the same extent as that observed in the hyperglycemic WT mouse hearts.

We subjected the MRL and control B6 mouse strains to 12 weeks of high fat diet (HFD) or control diet (CD) to investigate the abilities of the MRL mouse hearts to metabolically adapt to HFD.

The up-regulation of nitrotyrosine in Gata4 +/− ventricles further confirmed our hypothesis that reduced GATA4 levels hypersensitize mice hearts to imatinib-induced mitochondrial stress (Supplementary material online, Figure S4).

Furthermore, the response to iPOST in the ageing mouse heart seems to depend on the protocol of iPOST used, and a deficiency in STAT-3 associated with ageing might contribute to the dampened response (Boengler et al., 2008).

We attempted to detect mutant PPARγ allele in heterozygous mouse heart samples to confirm the presence of mutated allele.

Force's team then showed that Gleevec caused mouse hearts and cultured mouse heart cells to develop similar abnormalities.