Sentence examples for mouse have provided from inspiring English sources
Empirical studies on the ecology of the wood mouse have provided sufficient information for the species to be modelled mechanistically.
Gain- and loss-of-function strategies using transgenic over-expression and targeted ablation of candidate genes in in the mouse have provided important mechanistic insights into cardiovascular development, physiology and disease.
Likewise, results from experimental studies with genetically engineered model organisms, such as the fruit fly and the mouse, have provided the basis for the design of new clinical applications.
Analysis of genetically altered mice has revealed that mutations in other members of the SHH and Nodal signaling pathways also result in HPE phenotypes.Studies of HPE in the mouse have provided a framework for understanding key developmental events in human brain development and may provide new candidate genes for human HPE.
Recent studies in the mouse have provided additional important insight into the specific actions of R-spondin1.
Nearly three decades of research on the dystrophin-deficient mouse have provided valuable insights into the biochemical function of dystrophin.
The ability to model cancer in the mouse has provided a robust methodology to dissect the molecular etiology of cancer.
Study of the adbn−/− mouse has provided insight into the pathological factors that contribute to the development of muscular dystrophy.
Research on autoimmune mouse models, including the MRL+/+ lupus mouse, has provided strong and consistent support for a role of TCE; this has included studies of exposures at environmentally relevant concentrations through multiple routes (inhalational, dermal, and oral).
Although studies have been conducted in various model organisms, the mouse has provided an excellent model system owing to the similarity of its anatomy, physiology and genetics to the human.
To date, studies using genetically engineered mice have provided some information on subtype-related functions of the CNS α1-ARs.
