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On average, each mouse group exhibited nearly twice the number of group-specific copy number gains versus losses.
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Both mouse groups exhibited CT nerve responses to the sugars, although those of B6 mice were stronger.
Both mouse groups exhibited a CPIR following oral administration of 1 M glucose and 1 M sucrose but not 1 M fructose or water alone.
Likewise, the lung tissues of all treated mice groups exhibited no obvious morphological and histological differences when compared with the control group.
Starting from day 14, mice in this group exhibited a camel-like back and crooked spine.
In agreement with previous report [13], p73−/− mice in our group exhibited partially penetrant congenital hydrocephalus, and 20% of them died at an early age from intraventricular hemorrhage (Fig. 1A).
Most 0ΔNLS- and MS-immunized mice survived without any overt symptoms of disease for 30 days after vaginal challenge; however, 1 of 15 mice in each group exhibited limited perivaginal fur loss between 10 and 30 days after HSV-2 vaginal challenge.
VBP15 treated mice showed reduced calcification upon quantitation, with only 1 2 mice per group exhibiting any calcified fibres (n = 6 mice/group).
Monotherapy treatment with F16 IL2 led to a minor tumour growth retardation, compared with the control group of mice treated with saline 10% DMSO, whereas all mice in the temozolomide group exhibited a strong tumour regression by day 30.
In contrast, in 9 out of 12 mice from the EAlow group, BUN was within normal range and none of the mice in the EAhigh group exhibited elevated BUN.
As shown in Figure 5A, diabetic nu/nu mice in the vehicle group exhibited stable euglycemia after islet transplantation until the removal of the graft-bearing kidney.
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