Consistent with our findings, it has been reported that in mouse liver, male gene signatures are enriched for functions such as immune response while female signatures are enriched in functions such as oxidoreductase activity and mitochondrion [ 75].
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The hypothalamus controls a number of important physiological functions, such as feeding and drinking, that are also abnormal in R6/2 mice [32], [33].
In the normal mice, the introduced cells had migrated to the hippocampus and cortex, areas critical for cognitive functions such as learning and memory.
Cardiac functions such as FS, SV, EF, and CO obtained for WT controls, eNOS−/− mice, and BM-transplanted eNOS−/− mice were not significantly different.
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We do not exclude the possibility that unexamined physical functions, such as olfaction, might be altered by NRG1 overexpression and influence social interaction scores of the Tg mice.
Spontaneous development of dacryoadenitis and keratoconjunctivitis is observed in mouse strains with defective T-reg function, such as the CD25KO strain used in these experiments, as well as the TGF-β1KO.
Indeed, polycystic disease can be caused in mouse models by homozygous loss-of-function mutations in proteins required for cilia formation or function, such as TG737, Kif3a, fibrocystin, and cystin [35], [36], [37], [38].
The PP2A activity is important for Emil2, which is required for maintenance of CSF arrest in frog eggs, [24], [25], while in mouse oocytes, such a function is not reported.
